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Long-term prenatal stress increases susceptibility of N-methyl-D-aspartic acid-induced spasms in infant rats |
Hyeok Hee Kwon, Taekwan Lee, Jinpyo Hong, Dong Woon Kim, Joon Won Kang |
Clin Exp Pediatr. 2018;61(5):150-155. Published online May 28, 2018 DOI: https://doi.org/10.3345/kjp.2018.61.5.150 |
Long-term prenatal stress increases susceptibility of N-methyl-D-aspartic acid-induced spasms in infant rats Prenatal stress promotes development of spasms in infant rats Late phase of long-term potentiation induced by co-application of N-methyl-d-aspartic acid and the antagonist of NR2B-containing N-methyl-d-aspartic acid receptors in rat hippocampus S2001 Prenatal Exposure to a Methyl-Donor Diet Increases Colitis Susceptibility in Infant Mice Audiogenic Seizure Susceptibility Is Induced by Termination of Continuous Infusion of γ-Aminobutyric Acid or an N-Methyl-d-aspartic Acid Antagonist into the Inferior Colliculus Model of infantile spasms induced byN-methyl-D-aspartic acid in prenatally impaired brain Glycine modulates N-methyl-D-aspartic acid induced learning facilitation in rats N-Methyl-d-aspartic acid-receptor antagonists block morphine-induced conditioned place preference in rats Long‐term Changes in Postnatal Susceptibility to Pilocarpine‐induced Seizures in Rats Exposed to Gamma Radiation at Different Stages of Prenatal Development Selegiline reduces N-methyl-D-aspartic acid induced perturbation of neurotransmission but it leaves NMDA receptor dependent long-term potentiation intact in the hippocampus |